Abstract
pylorL In cot~,tnlst, TGF-r was hmnd in the CM from cocuhure cells in the absence of PMA. Furthermore, TGFqa was also found in CM obtained from gastnc biopsy speciniens without PMA treatment. TGF-~* levels in the CM were correlated with scm~s {or activity and inflammation ot gastrius and the presence ot H. pylori. Conclusion: H pylon, but not IU113, might induce ectodomain shedding of TGF-~x in gastric epithelial cells.
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