Abstract
Pathomorphological changes in a focus of ischemia, the level of lipid peroxidation (as indicated by the content of products reacting with thiobarbituric acid), and the postischemic levels of adenyl nucleotides and tissue lipids were studied in rats with focal compression ischemia of the cerebral cortex. An increased level of the TBA-reactive products paralleled by a reduction of the concentrations of adenyl nucleotides and tissue lipids was found to be in clear-cut correlation with the processes of neuronal injury and death and with subsequent repair phenomena in the focus of cortical ischemia.
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