Abstract
Background and Aims : Previous studies indicate the participation of LIGHT(TNFSF14)-lymphotoxin beta receptor(LTβR) in atheroma progression. In apolipoproteinE-deficient mice (Apoe-/-), LTβR deficiency diminishes atheroma plaque, thought its absence in vascular smooth muscle cells(VSMCs) aggravates atheroma altering artery tertiary lymphoid organ (ATLO). In this research, the effect of whole body and hematopoietic stem cell (HSC) specific gene inactivation of Light gene in Apoe-/- mouse model was investigated.
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Published version (
Free)
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
