Abstract
Mammals have lost the ability to replace damaged sensory hair cells of the inner ear through the proliferation of remaining hair cells. In a Perspective, [Taylor and Forge][1] discuss exciting new work in mice that identifies the retinoblastoma protein as a key regulator of the nonproliferative state of inner ear hair cells ([ Sage et al .][2]). They postulate that blocking expression of the retinoblastoma protein may form the basis of therapies that could help to treat hearing impairments by coaxing hair cells to proliferate. [1]: http://www.sciencemag.org/cgi/content/full/307/5712/1056 [2]: http://www.sciencemag.org/cgi/content/short/307/5712/1114
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