Abstract
Adverse posttraumatic neuropsychiatric sequelae (APNS) are common among trauma survivors. Traditional classification of APNS (e.g., posttraumatic stress, pain) artificially fragment APNS into syndromes based on medical specialty bailiwick, unmoored to brain function. Mapping trajectories of discrete symptom types over time, assessing biobehavioral markers of these trajectories, and evaluating how these trajectories developmentally interact and aggregate may provide new insights into APNS pathogenesis and/or treatment.
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