Abstract
ARDS is a life-threatening disease of multifactorial etiology caused by a direct or indirect insult to the pulmonary lobules. We have tested a therapeutic intervention on a previously defined model of ARDS,1 in which the three fundamental elements of a disease process2—pathogenesis, structural alterations, and functional consequences—were described during the course of the disease. In this single “hit” model, degree and duration of the host defense response (HDR) determine the adaptive vs maladaptive evolution of the reparative process and final outcome.
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