Abstract

Monaural phase discrimination was evaluated at 1000 Hz in six normal-hearing listeners as a function of the frequency difference between components in three-tone complexes at 40, 60, 80, and 100 dB SPL. The phase of the center component of 100% sinusoidally amplitude-modulated (SAM) waveforms was shifted by 90 degrees to produce quasi-frequency-modulated (QFM) waveforms that had identical long-term power spectra to the SAM waveforms but with different amplitude envelopes and temporal fine structure. At low modulation frequencies, where spectral components were close together and presumably all well within a single auditory filter, normal-hearing listeners could easily discriminate QFM from SAM waveforms. As modulation frequency increased, a point was reached where listeners could no longer distinguish QFM from SAM waveforms, referred to here as the critical bandwidth for phase discrimination (CBphs). Discrimination performance (d') was measured as a function of modulation frequency to yield a psychometric function for phase discrimination. From the psychometric function, CBphs was defined as the modulation frequency corresponding to d' = 1.0. At a carrier frequency of 1000 Hz, CBphs increased with level between 40 and 80 dB SPL according to the relation: CBphs = 34.I(0.136). Above 80 dB SPL, very little change in CBphs with level was seen. The increase with level of the geometric mean CBphs was predicted from level-dependent auditory filter slopes inferred from forward-masked tuning curves, as was the tendency to reach an asymptote above 80 dB SPL. Comparisons with previous work indicate that CBphs values from the best performing subjects were well within the audibility region for cubic difference tones. It is proposed that internally generated cubic difference tones interact with externally generated acoustic components, both limited by a level-dependent auditory filter, to produce an internal excitation envelope that is the basis for discriminating between SAM and QFM waveforms. It is also suggested that individual differences at low levels may be due to internal phase ambiguities.

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