Abstract
Pessi et al1 showed in 101 cases of ST-segment elevation myocardial infarction that bacterial DNA typical for endodontic infection was 16 times higher in thrombi than that found in patients’ blood samples. Oral viridans streptococci was measured in 78% of thrombi and periodontal pathogens in 35%.1 Furthermore, there was a significant association between the presence of periapical abscess and endodontic bacteria in the thrombus aspirates (odds ratio, 7.71) in 30 patients with available dental panoramic tomography. This is an exciting finding that begs the question of how the presence of a chronic oral infection can mediate an acute transition to plaque thrombosis. First, it underscores …
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