Abstract
To understand the leptin's action on hypothalamic arcuate nucleus (ARC), Ca 2+/calmodulin (CaM) -dependent response in leptin-treated rat ARC was investigated. The in vitro phosphorylation of ARC extracts from leptin-treated rats did not respond to CaM. Although CaM-dependent protein kinase II activity was not affected by leptin, the gene expression of calcineurin, CaM-dependent phosphatase, increased in leptin-treated rats.
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