Abstract

Left ventricular hypertrabeculation (LVHT), also known as noncompaction, is an abnormality of the left ventricular myocardium, characterised by separation of the left ventricular wall into a compacted, thin outer layer and a thicker, crenated, inner layer, predominantly of the apex and the lateral wall, distally to the papillary muscles [1]. LVHT is frequently associated with other cardiac abnormalities, chromosomal abnormalities, or neuromuscular disorders [2]. LVHT patients carry an increased risk to develop malignant arrhythmias [3], heart failure [4], or possibly peripheral embolism.

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