Abstract
Thrombotic events in neonates may origin from fetal life. A 4-day-old newborn infant with a family history of heterozygous type 1 protein C deficiency was diagnosed with left lung hypoplasia and left pulmonary artery thrombosis. Its source was prenatally closed ductus arteriosus. Surgical removal of the thrombus was performed.
Highlights
Thrombotic events in neonates have been recorded with increasing frequency, especially when an inherited thrombophilia is present [1]
We present the case of a newborn infant with pulmonary artery thrombosis (PAT) and left lung hypoplasia with a family history of heterozygous type 1 protein C deficiency
No left pulmonary artery (LPA) lumen and flow could be identified; the whole LPA lumen was completely filled with a homogeneous content with increased echogenicity
Summary
Thrombotic events in neonates have been recorded with increasing frequency, especially when an inherited thrombophilia is present [1]. We present the case of a newborn infant with pulmonary artery thrombosis (PAT) and left lung hypoplasia with a family history of heterozygous type 1 protein C deficiency. In the course of several days the mass further extended into the RPA, with further increase of the pressure gradient. Surgical removal of this pulmonary artery mass was performed. A white colored matter of atheromatous appearance originating from the ductal orifice filled the LPA and extended into the RPA. This white matter was removed; the periphery of the hypoplastic LPA remained obstructed.
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