Abstract
0.14± 0.17mcg/L, p< 0.0001) increased post-race and correlated with decreased RVEF (r= 0.52, p= 0.001 and r= 0.48, p= 0.003 respectively). All measures of RV function decreased post-race whilst LV measures did not change (see table). Changes in RVEF, RV SRs and BNP all increased with race duration (p< 0.05), weekly training hours (22± 5 vs. 15± 5 h, p= 0.001) and VO2max (60.0± 5.7 vs. 55.8± 4.4ml/min/kg, p= 0.03). At baseline, abnormal septal DGE was identified in 5/39 athletes and was associated with lower RVEF (47.1± 5.9% vs. 51.1± 3.7%, p= 0.042). Conclusions: Post-race biochemical abnormalities were explained byRV (andnot LV) dysfunctionwhich increased with race duration and were not reduced by training. Localised myocardial fibrosis was seen in some athletes at baseline and associated with lower RVEF suggesting the possibility of longer term myocardial damage.
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