Late-Developing Supernumerary Premolars: Analysis of Different Therapeutic Approaches.

Sergio Paduano,Domenico Aiello,Ambrosina Michelotti,Cristina Grippaudo,Alessandra Lucchese,Roberto Rongo

doi:10.1155/2016/2020489

Abstract

This case series describes the different potential approaches to late-developing supernumerary premolars (LDSP). LDSP are supernumerary teeth (ST) formed after the eruption of the permanent dentition; usually they develop in the premolar region of the upper and lower jaw. The choice to extract or to monitor the LDSP depends on many factors and has to be carefully planned due to the several risks that either the monitoring or the extraction could provoke. These four cases of LDSP showed different treatment plan alternatives derived from a scrupulous assessment of the clinical and radiographic information.

Highlights

Supernumerary teeth (ST) or hyperodontia is one of the less frequent developmental anomalies characterized by an excess number of teeth with respect to the usual configuration of 20 deciduous and 32 permanent
Several cases of late-developing supernumerary premolars (LDSP) are discussed in the literature [12, 18,19,20,21,22,23]; even if uncommon, this condition has to be taken into account in the different phases of an orthodontic treatment
The cases highlight the late development of supernumerary teeth in the premolar region at different times with respect to the orthodontic treatment

Summary

Introduction

Supernumerary teeth (ST) or hyperodontia is one of the less frequent developmental anomalies characterized by an excess number of teeth with respect to the usual configuration of 20 deciduous and 32 permanent. The prevalence of ST varies between 0.1% and 3.8% [1]; they are more reported in the permanent dentition (1–3% of general population) than in primary dentition (0.8% of population) [2]. Several theories were proposed to explain this condition such as the phylogenetic theory [3], the dichotomy theory (splitting of the tooth germ) [4], and the hyperactivity theory (hyperactive dental lamina) [5]; it is most likely to be due to a combination of genetic and environmental effects [6]. Certain genetic related syndrome, cleidocranial dysplasia, Gardner’s syndrome [8, 9], and cleft lip and palate [10] predispose to ST

Results

Discussion

Conclusion