Abstract
Cervical spondylotic myelopathy (CSM) is the most common cause of spinal cord dysfunction in the world. The prevalence of CSM has never been measured, but is estimated to be 1.6 per 100,000 inhabitants. It is frequently associated with degenerative changes (usually superimposed on a congenitally stenotic canal), which result in compression of the cervical spinal cord. There is universal agreement that compression of the spinal cord as the primary pathophysiologic mechanism is responsible for myelopathy; however, the neurologic dysfunction is almost certainly magnified by motion. In most patients, the combination of static compression of a congenitally stenotic canal with dynamic factors leads to the development of myelopathy. It is generally accepted that in the pathophysiology of CSM.
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