Abstract
The conventional stimulants of gastric secretion (histamine, pentagastrin, and insulin) results in a fall in lactic acid concentration and usually a rise in lactic acid output. Agents that impose indirectly or directly a lactic acid load (glucose, fructose, xylitol, epinephrine) usually result in an increase in lactic acid concentration and output. It is considered that non-ionic diffusion plays little role in lactic acid secretion, and that the lactic acid content of gastric juice reflects the metabolic processes in the gastric mucosal cells.
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