Abstract
Adverse cutaneous reaction to anti-tuberculosis drugs (ATD), such as maculopapular eruption (MPE), is one of the most common cause of discontinuation of scheduled treatment of tuberculosis (TB). Tumor necrosis factor-α (TNF-α) plays an important role in drug- or drug metabolite-induced immune responses and we recently reported that TNF-α genetic polymorphism -308G/A is significantly associated with ATD-induced hepatitis. In this study, we aimed to investigate associations between TNF-α -308G/A and ATD-induced MPE.
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