Abstract
22-year-old male patient with no heart disease, who was given an ambulatory medication with analgesics due to an acute renal crisis. After the drug administration, the patient presented dyspnea, cyanosis, and hemoptysis. There was suspicion of anaphylactic shock, which was treated, but there was no improvement in the clinical condition. The patient was referred to the Intensive Care Unit, where tests were performed showing elevated cardiac enzymes and Immunoglobulin E and Computed Tomography of Thoracic revealed alveolar hemorrhage. He [...]
Highlights
Kounis syndrome is consisted of an acute coronary syndrome but despite the usual atherosclerosis etiology has its origin in anaphylactic or anaphylactoid allergic reactions
The syndrome is caused by inflammatory mediators such as histamine, platelet activating factor, arachidonic acid products, neutral proteases, cytokines, and chemokines that trigger a cascade that results in the activation of inflammatory cells
This set of mediators, in turn, feed a vicious cycle with the release of more mediators. All of this leads to coronary vasospasm justifying its clinic of an acute coronary syndrome.[3]
Summary
Kounis syndrome (referred here as SK) is consisted of an acute coronary syndrome but despite the usual atherosclerosis etiology has its origin in anaphylactic or anaphylactoid allergic reactions. The syndrome is caused by inflammatory mediators such as histamine, platelet activating factor, arachidonic acid products, neutral proteases, cytokines, and chemokines that trigger a cascade that results in the activation of inflammatory cells. This set of mediators, in turn, feed a vicious cycle with the release of more mediators. Pereira et al Kounis Syndrome complaining of severe low back pain on the right associated with nausea and vomiting He reported being under treatment for epilepsy. The patient evolved into sepsis and cardiorespiratory arrest and did not answer to any resuscitation maneuvers He died after 19 days of hospitalization
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