Abstract
Hyperphosphatemia is a well-known feature of advanced chronic kidney disease (CKD) and a major risk factor for cardiovascular events in patients with CKD, inducing calcium phosphate supersaturation and vascular calcifications.1 Although the serum phosphate level is not recognized as an independent risk factor for CKD progression, increased urinary phosphate excretion has been associated with CKD progression in small cohort studies.2,3 In experimental studies, exposure to a high-phosphate diet has been shown to accelerate kidney damage in rats with reduced nephron number induced by partial nephrectomy.
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