Abstract
Tinea capitis is one of the most frequent dermatophytic infections of the scalp in children. The most common pathogen is Trichophyton torsurans and Microsporum canis. One of the most severe forms is described as kerion, which usually occurs due to advanced infection and exaggerated host response resulting in widespread inflammation, frequently with pustule and abscess formation. Prolonged infection may lead to permanent alopecia. Differential diagnosis and adequate treatment play critical role especially when it affects the children.
Highlights
Tinea capitis is one of the most frequent dermatophytic infections of the scalp in children [1]
Clinical spectrum of symptoms varies from very moderate changes with a slight seborrheic dermatitis to very severe forms with intensive inflammation and large plaques of alopecia [3]
One of the most severe forms described in the literature is known as kerion [3]
Summary
Tinea capitis is one of the most frequent dermatophytic infections of the scalp in children [1]. Infection may lead to the hair damage due to its endothrix pattern infection [2] This way of fungal growth results in hair breakage and "black dot" tinea capitis. Microsporum spp. infection with its ectothrix pattern usually leads to dry, clay patches of alopecia. Clinical spectrum of symptoms varies from very moderate changes with a slight seborrheic dermatitis to very severe forms with intensive inflammation and large plaques of alopecia [3]. One of the most severe forms described in the literature is known as kerion [3] It is a state, where advanced fungal infection and excessive host response are present [3, 4]. Especially with large plaques of alopecia and excessive inflammation an oral treatment should be introduced (Tab. 1)
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