Jodom-izazvan hipertireoidizam - da li ste?

Ilker Sengul,Anton Pelikán,Demet Sengul

doi:10.24125/sanamed.v15i2.458

Abstract

Iodine can be obtained by consumption of the foods that include it or to which it is added. Iodine-induced hyperthyroidism, also known as Jod-Basedow Syndrome, usually rise in the cases of underlying thyroid disease, such as autoimmune thyroid disease, latent Graves disease, non-toxic diffuse or nodular goiter or previous thyroid surgery. Iodine-induced hyperthyroidism is frequently self-limited with a period of 1-18 months after the discontinuanceation of iodine supply/replation. The prognosis is frequently favorable, the majority returns to the baseline hormonal status. The long-term outcomes of Jod-Basedow effects remain unknown due to the scarcity of phenomenon. Nonetheless, it is remarkable that treatment, devoted to the underlying thyroid diseases, should be addressed after resolution of the acute episode. Furthermore, an interprofessional health care team must serve and officiate not only to treat iodine-induced hyperthyroidism, but also to concern themselves actively in prophylaxis.

Highlights

Iodine can be obtained by consumption of the foods that involve it or to which it is annexed
The prevalence of hyperthyroidism that has been reported subsequent to the iodine administration is 1-20%, while the higher frequency, 10-20%, appears in the individuals with nodular goiter living in the areas of iodine deficiency [2, 3]
The autonomy of underlying areas within the thyroid gland leads to thyrotoxicosis and the autonomous areas generate the thyroid hormones independently of normal regulatory mechanisms as exogenonous iodine supply is augmented usually by iodinated contrast media used in conjunction with computed tomography scans, angiography, and various other imaging studies, or by intake of iodinated antiseptic solutions, and oral supplements [5, 9]

Summary

Introduction

Iodine can be obtained by consumption of the foods that involve it or to which it is annexed. The recommended minimum daily intake of iodine is 150 mg for nonpregnant adults, > 12 years; 220 to 250 mg for pregnants; 290 mg for lactating women, and 90 mg to 120 mg for children, 0-59 months and 6-12 years, respectively [1]. The prevalence of hyperthyroidism that has been reported subsequent to the iodine administration is 1-20%, while the higher frequency, 10-20%, appears in the individuals with nodular goiter living in the areas of iodine deficiency [2, 3]. Iodine-induced hyperthyroidism, known as Jod-Basedow Syndrome, scarcely occurs in cases in the absence of an underlying thyroid disease like autoimmune thyroid disease, previous thyroid surgery, latent Graves disease, and nontoxic diffuse or nodular goiter [4,5,6,7,8].

Results

Conclusion