Abstract
Mechanisms of autoimmunity in primary immunodeficiencies (PID) are, in many situations, unclear. Despite being linked with many PIDs (namely humoral), juvenile idiopathic arthritis (JIA) has rarely been associated with DNA-repair disorders, such as Ataxia-telangiectasia (A-T).
Highlights
Mechanisms of autoimmunity in primary immunodeficiencies (PID) are, in many situations, unclear
We report a case of juvenile idiopathic arthritis (JIA)-like in a male patient with A-T
A 3 year-old boy with A-T presented with pain and swelling of fourth metatarsophalangeal (MTP) joint of left foot
Summary
Mechanisms of autoimmunity in primary immunodeficiencies (PID) are, in many situations, unclear. Filipa Furtado1, Ana Isabel Cordeiro2, João Neves2, Marta Conde1* From 21st European Pediatric Rheumatology (PReS) Congress Belgrade, Serbia. Introduction Mechanisms of autoimmunity in primary immunodeficiencies (PID) are, in many situations, unclear.
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