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Abstract
Pacemaker activity of the heart involves interplay between several ionic currents that ins uence spontaneous diastolic depolarization of the sinoatrial node, including I f current. The search for pure heart rate-lowering agents to prevent angina without over all adverse effects of β-blockers [1] began more than three decades ago. The discovery of I f current and I f channels offered a possible approach to the developing pure heart rate-lowering agents. [1]
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