Abstract
A number ofAspergillus nidulans mutants unable to grow on lactose or growing very poorly on this sugar have been isolated. They may be divided into two major groups: to the first belong mutants in which β-galactosidase can be induced by galactose but not by lactose. Mutants of the second group are induced neither by lactose nor by galactose. Mutants of the first group showed an impaired lactose-permease system, while those of the second group most likely concern β-galactosidase structural or regulatory genes as they show a normal rate of lactose uptake. Genetic analysis revealed that mutants from the first group fall into three different loci and those from the second into four loci. No mutant has been found so far with the lactose-permease system and β-galactosidase simultaneously impaired, or with a constitutive level of either activity.
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