Abstract

Ischemia-reperfusion damage is a complex pathological process that begins with tissue anoxia and continues with the production of free oxygen radicals, expanding with the inflammatory response. The literature suggests the importance of antioxidant and anti-inflammatory treatment to treat ischemia-reperfusion-related tissue damage.

Highlights

  • Ischemia-reperfusion damage is a complex pathological process that begins with tissue anoxia and continues with the production of free oxygen radicals, expanding with the inflammatory response

  • Healthy cellular functions are maintained through aerobic metabolism, whereas anaerobic metabolism is observed in ischemic cells [1]

  • Adenosine triphosphate (ATP) produced in healthy tissues is lysed into adenosine monophosphate (AMP), adenosine, inosine and hypoxanthine

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Summary

Introduction

Ischemia-reperfusion damage is a complex pathological process that begins with tissue anoxia and continues with the production of free oxygen radicals, expanding with the inflammatory response. İskemi-reperfüzyon hasarı, dokunun oksijensiz kalması ile başlayan, serbest oksijen radikallerinin üretimi ile devam eden ve inflamatuar yanıtla genişleyen karmaşık patolojik bir süreçtir. Ischemic Damage Ischemia is defined as an asphyxiated tissue state as a result of reduced or absent blood flow.

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