Abstract
Introduction: Ischemia and reperfusion (I/R) following cardiac surgery regularly elicits an inflammatory response with excessive release of oxygen species that is frequently associated with morbidity and mortality. Macrophage migration inhibitory factor (MIF) is a pro-inflammatory/pleiotropic cytokine that might compensate oxidative stress and thus counteract the various detrimental effects after myocardial I/R. We hypothesized that intraoperative MIF secretion might be due to I/R and enhance antioxidative capacity in patients during cardiac surgery.
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