Abstract
Background The inclusion of limited joint mobility (LJM) as a risk factor for plantar neuropathic foot ulceration in diabetes is interspersed throughout the literature. This is commonly believed to occur through connective tissue thickening and stiffening, thereby reducing available static and dynamic motion below that required for normal foot function. High underfoot pressures are postulated to result, leading to increased ulcer risk. This project investigated this theory as it relates to ankle joint dorsiflexion in people with a range of lower limb complications due to diabetes.
Highlights
The inclusion of limited joint mobility (LJM) as a risk factor for plantar neuropathic foot ulceration in diabetes is interspersed throughout the literature
This is commonly believed to occur through connective tissue thickening and stiffening, thereby reducing available static and dynamic motion below that required for normal foot function
To the static measures was the trend for mean dynamic foot dorsiflexion used in gait to be approximately 3° to 4° greater in the DNU group compared to the DNC group
Summary
Is there a relationship between static and dynamic ankle joint dorsiflexion during gait in people with a history of neuropathic ulceration? Background The inclusion of limited joint mobility (LJM) as a risk factor for plantar neuropathic foot ulceration in diabetes is interspersed throughout the literature. This is commonly believed to occur through connective tissue thickening and stiffening, thereby reducing available static and dynamic motion below that required for normal foot function.
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