Abstract
In a recent issue of Trends in Plant Science, Andrew J. Love et al. [1] provided an interesting point of view regarding regulatory overlap between apoptotic-like and autophagic programmed cell death (PCD). Taking into account that the two forms of cell death share cytological and biochemical features, the authors reasonably hypothesized that they might also possess a common set of molecular regulators. The authors further postulated that the nature of the stimuli would determine the cell death rate and, consequently, whether apoptotic-like (rapid) or autophagic (slow) PCD would be engaged.
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