Abstract
With the pathogenesis of Alzheimer's disease (AD) continuing to elude researchers, new approaches that look beyond amyloid β plaques (Aβ) deposition and tauopathy are needed to broaden the understanding of neurodegenerative mechanisms that propagate dementia.
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Published version (
Free)
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
