Abstract
Since anthracyclines are still among the most frequently used antineoplastic drugs, early detection and/or prediction of anthracycline-induced cardiotoxicity is urgently needed. Serum levels of cardiac troponin T (cTnT) are increasingly becoming recognized as potential biochemical markers of even subclinical myocardial injury. In a recent paper, Lipshultz et al. [1] affirmed that ‘the serum level of cardiac troponin T is an accurate surrogate for acute myocardial injury in children, specifically that related to doxorubicin’.
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