Abstract
Programmed cell death (PCD) is a multifaceted process involved in cell number control, removal of diseased or inflamated cells and maintaining homeostasis between dying and newborn cells. It is a fine regimented process under strict genetic control. The process is not only associated with developmental programs of plants and animals but also observed during extreme fluctuations in environmental factors, as well as during noncompatible biotic interactions. In particular, stress-induced PCD in plants has been hypothesized as one of the survival strategies. At the regulatory stages of the process, amplification of cellular reactive species of oxygen (ROS) and nitrogen (RNS) acts as key signalling events for execution of PCD. Although roles of ROS and RNS in execution of PCD have been well studied independently, information about cross-talks between ROS and RNS are limited. In this chapter, efforts have been made to compile the available information regarding involvement of ROS, RNS and their cross-talk during the execution of PCD in plants.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
