Abstract
Electromagnetic screening for several days induced triphasic changes in nociception in mollusks and mice: an initial phase of hyperalgesia was followed by an antinociceptive phase and subsequent normalization of measures of nociception. In mollusks, these changes developed more slowly and the hyperalgesia phase was more marked than in mice. Daily administration of melatonin to the animals eliminated the screening-induced hyperalgesia and induced an earlier and more marked antinociceptive effect. These changes appeared to be linked with initial suppression of melatonin secretion by electromagnetic screening, resulting in hyperalgesia. Melatonin secretion then increased, which was apparent as an increase in the antinociceptive effect.
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