Abstract
Background: Kuru plaque and type i protease-resistant prion protein (PrP) in Creutzfeldt-Jakob disease (CJD) patients with methionine homozygosity at codon 129 of PrP gene (129MM), denoted as CJD-MMiK, are halmarks to identify acquired CJD cases caused by transmission of the V2 sporadic CJD (sCJD) strain to individuals with 129MM.
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