Abstract
Children infected with human immunodeficiency virus type 1 (HIV) are more likely to acquire infections with encapsulated bacterial pathogens of childhood than their non-HIV-infected peers. It has been shown thatStreptococcus pneumoniae (pneumococcus) accounts for 35% of the bacterial isolates from blood and cerebrospinal fluid in this group of children. We wished to learn what proportion (if any) of the incidence of community-acquired, invasive pneumococcal disease in HIV-infected children could be attributed to splenic dysfunction, as measured quantitatively by enumerating the number of pocked red blood cells in peripheral blood (Pearsonet al Pediatrics 1985; 76:392).
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