Abstract
Dopamine is often used for maintenance of haemodynamic stability in critically ill patients in the setting of intensive care units. ‘Renal’ doses (1 to 2 μg/kg/min) or ‘pressor’ doses (above 5 to 10 μg/kg/min) are used distinctly for various clinical disorders. Dopamine may be associated with diabetes insipidus, manifested by serum hypertonicity, urine hypotonicity and polyuria.[1] The mechanism of the potentiation of free water excretion remains obscure, although inhibition of pituitary vasopressin or increased solute excretion have been proposed.[1] In this case report, we describe a patient who developed acute diabetes insipidus associated with dopamine administration, where neither of these mechanisms appears likely.
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