Abstract
Objective: Ovarian cancer can evade immune responses by inducing endoplasmic reticulum stress in intratumoral leukocytes. In the ovarian tumor microenvironment, the endoplasmic reticulum stress-activated transcription factor X-box binding protein 1 (XBP1s) causes immune cell dysfunction and facilitates malignant progression. However, the clinical significance of XBP1s expression in ovarian cancer remains elusive. We sought to evaluate the association between intratumoral XBP1s levels and ovarian cancer patient outcomes.
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