Abstract
Abnormal return of cutaneous sensibility is common after bum injuries, with many patients complaining of neuropathic pain. Chronic neuropathic pain has been described in healed bums due to deficiencies in the reinnervation of the scar; abnormalities in newly regenerated nerve endings or possibly a more NMDA mediated central mechanism. Ketamine, a non-competitive NMDA receptor antagonist, has been used for analgesia in many cases of pain syndrome and it's routes of administration are variable. Blocking of the NMDA receptors abolishes the hypersensitivity in patients with neuropathic pain. Therefore, ketamine was tried in postburn neuropathic pain, with a similar antagonistic effect to that of a NMDA antagonist expected. A postburn neuropathic pain patient, who was successfully managed without any significant sign of side effects, using an intramuscular injection of ketamine, was experienced at our clinic.
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