Abstract
In the initial article 1 of this series, we described our methods of investigating intracranial hydrodynamics in human cadavers under experimental conditions that rendered certain of our results applicable to the living. Two of our conclusions were: The lateral ventricles and the cisternal and lumbar subarachnoid spaces remained in free communication under all experimental variations in pressure, provided no distortion of the subtentorial structures was artificially produced. In the absence of such distortion there was no evidence of tentorial, medullary or cerebellar movement under any experimental variations in subarachnoid fluid pressure. Specifically, we could not produce descent of the cerebellum or medulla into the foramen magnum by hydrodynamic means alone, e.g., sudden release of high intracranial fluid pressure through rapid lumbar or cisternal tap. We were therefore unable to confirm the generally accepted importance of the mechanical factor in the etiology of cerebellar herniation and bulbar compression in cases in
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