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Abstract
The pivotal role of intracellular free [Ca2+] fluctuations in the control of cellular functions such as contraction and secretion, including the release of neurotransmitters, was recognized many decades ago (see Rubin, 1982). More recently, the list of cellular functions triggered or modulated by the levels of Ca2+ i has grown enormously. Additional functions regulated by [Ca2+]i include neuronal excitability, synaptic plasticity, gene expression, cellular metabolism, cell division and differentiation, and programmed cell dead (Miller, 1991; Clapham, 1995). Paralleling the growth in this list of Ca2+-controlled functions, a multiplicity of cellular mechanisms aimed at maintaining resting free [Ca2+]i in the range of 100 nM for most cells has been described, allowing increases in Ca2 i levels that are specific in their magnitude, time course and spatial distribution, according to the cell function activated (Toescu, 1995).KeywordsCarotid BodyCatecholamine SecretionCarotid Sinus NerveArterial ChemoreceptorChemoreceptor CellThese keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.
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