Abstract

Introduction. In this paper, we aimed to present five Papaver rhoeas intoxication cases, which is very rare in the literature. Case 1. A 35-year-old female patient was admitted to our emergency room with the complaints of nausea, restlessness, and dyspnea developing 3 hours after eating Papaver rhoeas. On physical examination, her general condition was moderate; she was conscious and the vital findings were normal. The pupils were myotic. She was transferred to the toxicology intensive care unit as she experienced a generalized tonic clonic seizure lasting for three minutes. Case 2. A 41-year-old female patient was brought to our emergency room by 112 ambulance as she had contractions in her arms and legs, unconsciousness, and foam coming from her mouth two hours after Papaver rhoeas ingestion. On physical examination, she was confused, the pupils were myotic, and she was tachycardic. Arterial blood gases analysis revealed lactic acidosis. Case 3. A 38-year-old female patient was admitted to our emergency room with complaints of nausea and vomiting two hours after ingestion of Papaver rhoeas. Her physical examination and tests were normal. Case 4. A 34-year-old male patient was admitted to our emergency room with complaints of numbness and loss of power in his arms and legs one hour after Papaver rhoeas ingestion. He was hospitalized at the toxicology intensive care unit for follow-up and treatment. Dyspnea and bradycardia developed on the follow-up. The oxygen saturation without oxygen support was 90%. ECG revealed sinus bradycardia. The cardiac enzymes did not increase. Case 5. A 42-year-old female patient was brought to our emergency room by 112 ambulance with contractions in her arms and legs and unconsciousness two hours after Papaver rhoeas ingestion. On her physical examination, she was confused and the pupils were myotic. Arterial blood gases analysis revealed lactic acidosis. Conclusion. All patients were followed up for a few days and then discharged from the hospital with recovery. Unconscious consumption of Papaver rhoeas leads to a clinical condition resembling morphine intoxication, CNS depression, and epileptic seizures.

Highlights

  • We aimed to present five Papaver rhoeas intoxication cases, which is very rare in the literature

  • A 41-year-old female patient was brought to our emergency room by 112 ambulance as she had contractions in her arms and legs, unconsciousness, and foam coming from her mouth two hours after Papaver rhoeas ingestion

  • A 34-year-old male patient was admitted to our emergency room with complaints of numbness and loss of power in his arms and legs one hour after Papaver rhoeas ingestion

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Summary

Introduction

Papaver rhoeas is a plant which spontaneously grows in fields and grasslands and its flowers are in the shape of four blood red pots [1, Image 1]. Liver function test, renal function test, cardiac troponins, electrolytes, complete blood cell counts, electrocardiography (ECG), and computed tomography of the brain were normal. 250 grams has been eaten from baked Papaver rhoeas On physical examination, she was confused, the pupils were myotic, and she was tachycardic. Renal function test, cardiac troponins, electrolytes, complete blood cell counts, and ECG were normal. Renal function test, cardiac troponins, electrolytes, complete blood cell counts, electrocardiography (ECG), and computed tomography of the brain were normal. Computed tomography of the brain and liver function test, renal function test, cardiac troponins, electrolytes, and complete blood cell counts were normal. The patient was followed up for one day and discharged with recovery

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