Abstract
Previously we and others demonstrated that atopic mice are protected from lethal respiratory viral infections. In our Sendai virus (SeV) model, we found that atopic mice were protected from developing post-viral airway disease. Since IL-33 is a major mechanistic component of the House Dust Mite (HDM) atopy model, which we used in our studies, we hypothesized that IL-33 was important in providing protection from lethality and post-viral airway disease in atopic mice.
Published Version
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