Abstract
Background and aim. In potential coeliac disease (CD) despite the presence of disease-specific serum antibodies the architecture of the small intestinal mucosa is normal (Marsh T0 or T1). The potential CD condition is a good in vivo model to clarify the early immunological events leading to gluten-induced enteropathy. For this purpose, we analysed cytokines and transcription factors involved in T cell activation (interleukin (IL)-2), Th1 differentiation (interferon (IFN)-γ, Tbet, inteferon regulatory factor (IRF)-1) and immune regulation (IL-10).
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