Interactions Between Environmental Health and Pediatric Obesity

Abstract

Accumulating evidence suggests that air pollutant exposures affect multiple metabolic functions which may place populations at a higher risk for obesity. A pilot study was conducted to assess the feasibility of measuring personal exposures to fine particulate matter (PM2.5) and tobacco smoke, to assess the influence of these exposures on the risk of obesity in children in Kunming, a rapidly developing city in China.We recruited 52 children ages 4–5 years from three preschools in Kunming. Individual-level PM2.5 exposures were assessed using real-time personal monitors. Tobacco smoke exposures were assessed by a parent-reported questionnaire.Subjects' daily mean PM2.5 exposure (n = 52) was 32.8 (SD = 13.4) μg m−3 and daily tobacco exposure was 14.4 (SD = 21.1) cigarettes day−1. Our subjects’ daily mean exposure to PM2.5 was 31% higher than the level set by the WHO daily ambient concentration guidelines.We found personal PM2.5 monitoring to be a feasible method for measuring exposure to PM2.5 in young children. Future studies with larger sample sizes are warranted to explore the relationship between air pollution exposure and risk of obesity in China.