Abstract
Reactive Oxygen Species (ROS) in the bone environment can affect the behaviour of resident cells as well as the composition and morphology of the extra-cellular matrix. These alterations may reduce bone tissue performances, leading to a consequent collapse. Furthermore, there is a need for the creation of innovative biomaterials capable to counteract the increase of ROS produced by the fracture, the implantation process and the material itself, as well as by an age-related accumulation.
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