Abstract
Insulin resistance plays a central role in the pathophysiology of PCOS, and inositol phosphoglycan mediators have been shown to play a role in insulin transmembrane signaling. A deficiency of D-chiro-inositol phosphoglycan mediator at the tissue level has been studied as a marker of insulin resistance. These changes are also measurable in hemodialysate and urine. Thus, our objective is to study whether the administration of metformin to women with PCOS would alter the ratio of myo-inositol/D-chiro-inositolin serum and urine.
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