Abstract
During myocardial ischemia, ATP is broken down to the AMP-cata-bolites adenosine, inosine, (hypo)xanthine and uric acid. These apolar compounds can easily pass the cell membrane and enter the blood stream1. After reperfusion, energy charge and CrP are restored, but ATP levels remain low, because of purine loss2. To restore ATP levels, 3 main pathways exist: 1. adenosine phosphorylation, 2. inosine/hypoxanthine salvage, 3. de novo synthesis.
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Published version (
Free)
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
