Abstract
The immunopathogenesis of psoriasis involves the combined effect of predisposing genes and environmental risk factors, which excites the interaction between innate and adaptive immunity in the body. Recently, it has been gradually recognized that innate immune system takes part in the initiation of inflammation in psoriasis. Innate lymphoid cells (ILCs) mainly consist of 3 subsets, among which, natural cytotoxicity receptor-positive group 3 ILCs (NCR+ILC3) play a major role in innate immune responses in dermatitis. NCR+ILC3 can produce interleukin 17 (IL-17) and IL-22, and participate in the occurrence of psoriasis together with T cells. Key words: Psoriasis; Immunity; Interleukin-17; Interleukin-22; NCR+ ILC3
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