Abstract
Both 1-iodo-3-aminomethyl-5,6,7,8-tetrahydro-2-naphthol (ONO-3122), which increases endogenous PGH2, and PGH2 itself, significantly depressed vasopressin-induced osmotic water flow in the toad bladder. These results suggest that ONO-3122 increases endogenous PGH2 synthesis, and that PGH2 and/or its metabolites inhibit vasopressin-induced water flow.
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