Abstract
This study studied the adsorption of Lactobacillus fermentum CQPC10 (LF-CQPC10) to induce lead acetate. The animal experiments showed that LF-CQPC10 reduced SD rat serum, liver, kidney and brain lead and maintained the cellular structure and state of liver and kidney tissues. Furthermore, LF-CQPC10 could improve anti-inflammatory cytokinine by reducing anti-inflammatory cytokinine IL-1β, IL-6, TNF-α and IFN-γ and raising IL-10. At the same time, LF-CQPC10 can also enhance the δ-ALAD enzyme activity and decrease the enzyme activities of ATL and AST, and decrease the contents of serum BUN and CRE of rats with lead toxicity injury. Compared with EDTA, LF-CQPC10 not only has better lead ion degradation ability, but also has better anti-inflammatory and organ protection effects. As a lactic acid bacteria, which can be used in food industry, LF-CQPC10 reduce the lead content and lead toxicity in food.
Sign-in/Register to access full text options 
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
