Abstract
AbstractThe addition of ascorbic acid (20–160 μg) to mitochondrial preparations of rat or guinea pig liver has no effect upon the oxidation of [26‐14C]‐cholesterol to14CO2. The 7α‐hydroxylation of cholesterol by rat liver microsomes is also unaffected by addition of ascorbic acid. Hydroxylation by guinea pig liver microsomes is increased in the presence of ascorbic acid, but the results are not statistically significant.
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