Abstract
Nicotine injected intravenously in small doses (2.5–10.0 μg/kg) produced in anaesthetised cats a rise in cerebrospinal fluid pressure which was probably secondary to an increase in cerebral blood flow. Two possible mechanisms of action are discussed which involve the medullary vasomotor centre and brain stem reticular formation. It is suggested that the response is mediated via a sympathetic cerebral vasodilator pathway.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
